Non-Neoplastic Dermatopathology

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5 Disorders of the Subcutis

Figure 5.5.5 Lipodermatosclerosis. Septal fibrosis with hemosiderin deposition (siderophages).

Figure 5.5.6 Lipodermatosclerosis. Features of chronic venous stasis with thick-walled, grouped vessels in the superficial dermis that are associated with erythrocyte extravasation (arrow).

Figure 5.5.7 Morphea. “Squared-off” biopsy due to expansion of the dermis by sclerotic collagen bundles. The sclerosis extends into the subcutis with thickening of the septae. Perieccrine and deep perivascular inflammation that extends into the fat lobules.

Figure 5.5.8 Morphea. Sclerotic collagen bundles appearing swollen and with loss of usual fenestrations. The sclerosis encroaches upon the subcutaneous adipose tissue with obliteration of the fat. No appreciable lipomembranous or lipophagic fat necrosis.

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Figure 5.5.9 Morphea. Infiltrate of small lymphocytes and plasma cells within the deep dermis at the interface with the subcutis.

Figure 5.5.10 Morphea. Sclerosis replaces normal fat around eccrine unit and is accompanied by a lymphoplasmacytic infiltrate.